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Assessment of the cardiovascular effects of electroconvulsive therapy in individuals older than 50 years BJMBR
Takada,J.Y.; Solimene,M.C.; da Luz,P.L.; Grupi,C.J.; Giorgi,D.M.A.; Rigonatti,S.P.; Rumi,D.O.; Gowdak,L.H.W.; Ramires,J.A.F..
To evaluate the impact of electroconvulsive therapy on arterial blood pressure, heart rate, heart rate variability, and the occurrence of ischemia or arrhythmias, 38 (18 men) depressive patients free from systemic diseases, 50 to 83 years old (mean: 64.7 ± 8.6) underwent electroconvulsive therapy. All patients were studied with simultaneous 24-h ambulatory blood pressure and Holter monitoring, starting 18 h before and continuing for 3 h after electroconvulsive therapy. Blood pressure, heart rate, heart rate variability, arrhythmias, and ischemic episodes were recorded. Before each session of electroconvulsive therapy, blood pressure and heart rate were in the normal range; supraventricular ectopic beats occurred in all patients and ventricular ectopic...
Tipo: Info:eu-repo/semantics/article Palavras-chave: Electroconvulsive therapy; Blood pressure; Heart rate; Arrhythmia; Myocardial ischemia.
Ano: 2005 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2005000900009
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CIRBP protects H9C2 cells against myocardial ischemia through inhibition of NF-κB pathway BJMBR
Long,T.Y.; Jing,R.; Kuang,F.; Huang,L.; Qian,Z.X.; Yang,T.L..
Myocardial ischemia is a major cause of death and remains a disease with extremely deficient clinical therapies and a major problem worldwide. Cold inducible RNA-binding protein (CIRBP) is reported to be involved in multiple pathological processes, including myocardial ischemia. However, the molecular mechanisms of myocardial ischemia remain elusive. Here, we first overexpressed CIRBP by transfection of pc-CIRBP (pcDNA3.1 containing coding sequenced for CIRBP) and silenced CIRBP by transfection of small interfering RNA targeting CIRBP (siCIRBP). pcDNA3.1 and the negative control of siCIRBP (siNC) were transfected into H9C2 cells to act as controls. We then constructed a cell model of myocardial ischemia through culturing cells in serum-free medium with...
Tipo: Info:eu-repo/semantics/article Palavras-chave: Myocardial ischemia; CIRBP; NF-κB; Cell proliferation; Cell apoptosis.
Ano: 2017 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2017000400602
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Exercise may cause myocardial ischemia at the anaerobic threshold in cardiac rehabilitation programs BJMBR
Fuchs,A.R.C.N.; Meneghelo,R.S.; Stefanini,E.; De Paola,A.V.; Smanio,P.E.P.; Mastrocolla,L.E.; Ferraz,A.S.; Buglia,S.; Piegas,L.S.; Carvalho,A.A.C..
Myocardial ischemia may occur during an exercise session in cardiac rehabilitation programs. However, it has not been established whether it is elicited when exercise prescription is based on heart rate corresponding to the anaerobic threshold as measured by cardiopulmonary exercise testing. Our objective was to determine the incidence of myocardial ischemia in cardiac rehabilitation programs according to myocardial perfusion SPECT in exercise programs based on the anaerobic threshold. Thirty-nine patients (35 men and 4 women) diagnosed with coronary artery disease by coronary angiography and stress technetium-99m-sestamibi gated SPECT associated with a baseline cardiopulmonary exercise test were assessed. Ages ranged from 45 to 75 years. A second...
Tipo: Info:eu-repo/semantics/article Palavras-chave: Exercise test; Anaerobic threshold; Myocardial ischemia; Coronary artery disease; Technetium; 99mTc-sestamibi.
Ano: 2009 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2009000300008
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Ischemia and fibrosis: the risk mechanisms of hypertensive heart disease BJMBR
Frohlich,E.D..
Mechanisms underlying risk associated with hypertensive heart disease (HHD) and left ventricular hypertrophy (LVH) are discussed in this report and provide a rationale for understanding this very common and important cause of death from hypertension and its complications. Emphasized are impaired coronary hemodynamics, endothelial dysfunction, and ventricular fibrosis from increased collagen deposition intramurally and perivascularly. Each is exacerbated by aging and, perhaps, also by increased dietary salt intake. These functional and structural changes promote further endothelial dysfunction, altered coronary hemodynamics, and diastolic as well as systolic ventricular contractile function in HHD. The clinical endpoints of HHD include angina pectoris (with...
Tipo: Info:eu-repo/semantics/article Palavras-chave: Left ventricular hypertrophy; LVH; Risk factor; Myocardial ischemia; Ventricular fibrosis.
Ano: 2000 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2000000600010
Registros recuperados: 4
Primeira ... 1 ... Última
 

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